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Mutations of the interfacial residues in DET1 to glycine or alanine, including R488A/H485G, R488A/H485G/R306G/S312G or R488A/H485G/E380A/T382A reduced the interaction between DET1 and COP1

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Patients often experience persistent symptoms that are poorly correlated with thyroid hormone levels, an undefined treatment duration, and the need for long-term or definitive therapies

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