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This specific impairment of mitochondrial energy metabolism, combined with the downregulation of key cell cycle regulators such as c-Myc and Cyclin D1, provides a direct mechanistic explanation for the observed S-phase arrest and growth inhibition

Since this study was designed as a pilot trial with a short observation period (56 days), further studies are needed to clarify the long-term effect of a single or repeated oral FMT on cAD using a large population of dogs with mild to severe AD and appropriate controls

Paresthesias and gait disturbances may begin to improve within two weeks, although full recovery can take several months

Haematological responses occur within 12 weeks, but nerve repair is slower and may be incomplete if deficiency has been prolonged