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In the model proposed by Lee, homocysteine is shunted away from the methionine synthesis pathway and toward the GSH synthesis pathway, creating a relative deficiency of methyl donors in order to keep up with increased demand for the glutathione needed for POP conjugation ( Fig 3 )

Its potential biological significance in tissues where GLP-1(9-36) accumulates to measurable concentrations including cardiac and CNS tissue has been associated in the literature with cardioprotective and possibly neuroprotective effects

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