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A Multi-Pathway Model We can think of this as a convergence of several mechanisms: L-cell activation from viral or microbial sensing Vagal signaling altered by cytokine release Gut-brain axis dysregulation, mirroring GLP-1 agonist effects Delayed motility from enteric nervous system inflammation Together, these pathways could produce a temporary GLP-1-like state during and shortly after the viral illness

2015 Feb 6;12:7

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pylori infection include fecal antigen detection, urea breath detection and chemical staining of tissue biopsy (Shakir et al., 2023)

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