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First, the retrospective design and the reliance on an EHR-based database limited our results

It is well known that diabetes could lead to upregulation of intracellular reactive oxygen species (ROS), subsequently inducing inflammation, injury in endothelial cells and apoptotic cell death [4, 5]

Many endogenous peptides naturally exhibit multi-receptor activity, a feature that was historically viewed as a liability but may instead reflect evolutionary optimization for coordinated physiological control

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10.15252/embj.201696151 [DOI] [PMC free article] [PubMed] [Google Scholar] Zhang Z.Z., Lee E.E., Sudderth J., Yue Y., Zia A., Glass D., Deberardinis R.J., and Wang R.C