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As a hallmark of UC is neutrophilic infiltration and release of pre-formed MPO-containing granules, we examined MPO protein levels by immunohistochemistry and found significantly higher MPO levels in UC patient colons than control colons (Figure 1c)

While GHK has been shown to inhibit the inflammatory response and epithelial-mesenchymal transition (EMT) via the TGF-1/Smad 2/3 and IGF-1 pathways in bleomycin-induced fibrosis models [24], its broader anti-inflammatory effects in the context of pulmonary fibrosis pathogenesis may warrant further exploration

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