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Mechanistically, butyrate inhibits mTORC1, attenuates eIF2ATF4-dependent ISR signaling, and represses DGAT/ACSL-dependent LD biogenesis, whereas microglial Rptor overexpression abolishes these protective effects, identifying mTORC1 as an upstream metabolic checkpoint

ROS was visualised with using DCFH-DA, a non-specific redox probe that emits green fluorescence in the presence of any type of ROS

Biomaterials 212:3954

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