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Complementary clinical and meta-analytic data consistently demonstrate systemic redox imbalance, including lower glutathione reserves, altered GSH/GSSG ratios, increased lipid peroxidation, and dysregulated NRF2 signaling, linking oxidative status to immune activation and synaptic dysfunction (James et al., 2004

Hematol Oncol Clin North Am 2009 Jun;23(3):501-13, ix Available from: Reed JA, Shea CR

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3 Disruption of carnitine availability leads not only to impaired fatty acid metabolism, but also to clinical manifestations

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